لطفا صبر کنید ...
Exudative macular degeneration
The main cause of severe reduction of vision in macular degeneration is neovascularization of the sub-retinitis and associated exudative maculopathy. The serous material leaks from the lash and through the gaps in the brook membrane, causing a posterior deformation of the epithelium of the pigmentation. Of course, the most important histopathologic change that makes the patient prone to macular dysplasia and permanently drops the central vision is the creation of new vessels that grow back into the underlying region of the lash. In the early stages of neovascularization, the patient is asymptomatic, and even new arteries may not be detected by ophthalmoscopy or angiography. When a patient with genitourinary degeneration experiences a sudden reduction of central vision, subacute neovascularization should be suspected. If a fundus examination of the blood is found in the sub-retinal, exudate, or gray-laced lesions in the macular region, neovascularization is likely to occur, and an immediate fluorescein angiography is required to identify the lesion. However, some of the neovascular membranes are spontaneously improved; however, its somnolence is a permanent loss of central vision. The final stage of the cicatrians The exudative macular degeneration is a subcutaneous disco-oscillatory disorder, usually a subarachnoid dysfunction, usually in the center, leading to permanent loss of central vision.
Treatment
In the absence of neovascularization, the usefulness of medical treatment or surgical treatment of retinal pigmented epithelium septum necrosis has not been established. However, if a neovascular membrane exists underneath the outside of the fovea (200 micrometer or more than the center of the fovea-free area), laser-induced photocoagulation is indicated. In 50% of patients undergoing photocoagulation, within 2 years of neovascularization, recurrence of such patients will be necessary.